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The expression of these enzymes decreased by 75% at term and was barely detectable in preterm in labor patients or term labor patients [121] reiterating that NO has a role in maintenance of uterine quiescence

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Readers who want a broader orientation to the molecule can consult the What Is GHK-Cu pillar, which situates this systemic literature within the wider GHK-Cu picture

It is caused by mutations in the SLC22A5 gene, which provides information necessary for the synthesis of OCTN2, a protein responsible for carnitine transport into the cell