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Whereas, glucokinase expression and glycogen accumulation showed low glucose dose sensitivity

This supports the involvement of chronic inflammation and neuroinflammation as a potential mechanism that impairs physiological function, leading to the development and progression of the neurodegenerative disease (Vachatova et al

Research has found that GLPs regulate the microbial communities inside the mouth, intestines, pancreas, and lungs of rats suffering from T2DM by significantly increasing abundance of beneficial bacteria such as * Lactobacillus *, * Bifidobacterium *, * Prevotella *, * Blautia *, selenium-rich bacteria, along with * Coprococcus *, in addition to by inhibiting harmful bacteria such as * Staphylococcus *, * Escherichia *, * Ruminococcus torques (Holdeman and Moore 1974)*, * Streptococcus mutans (Clarke 1924)*, and *Clostridium perfringens (Veillon and Zuber 1898) * , consequently bettering glucose metabolism disturbances and alleviating reduced sensitivity to insulin ( However, the gut microbiotas composition determines how intestinal barrier functionality and directly impacts progression of diminished response to insulin and obesity