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Taken together, the data suggests that there is a vicious cycle of epithelial cell injury and immune activation, which in conjunction with increased susceptibility and abnormal repair mechanisms promote an abnormal airway epithelium in asthma ( 5.3 Epithelium-immune cell crosstalk in asthma In the asthmatic airway, alarmin and chemokine release induced cytokine activation is a well-recognised disease driving mechanism

It should be noted that there is statistically significant difference in the mean linear intercepts between mice exposed to smoke for 12 weeks and those for 24 weeks

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