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We modeled the age pattern of smoking risk on CVDs following the same steps we implemented for modeling doseresponse risk curves

CONCLUSION From the present study, we can concluded that continuous cigarette smoking increases erythrocyte count, hemoglobin concentration, hematocrit, leukocyte count, mean corpuscular volume and mean corpuscular hemoglobin concentration and these alterations might be associated with a greater risk for developing atherosclerosis, polycythaemiavera, chronic obstructive pulmonary disease and/or cardiovascular diseases

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