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Indeed, multiple studies have highlighted the importance of gap junctions in protecting cells against oxidative stress-induced cell death 25 and in modulation of cell proliferation and survival 25,26 , tumorigenesis 27 , and differentiation 28,29

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Unlike previously reported markers such as regulatory or secretory proteins, microRNAs, or gene mutations, which typically arise as secondary responses to mutagenesis 36 , CEdG is directly associated with DNA damage itself